Low-carb and ketogenic diets are popular among clinicians and patients, but the appropriateness of reducing carbohydrates intake in obese patients and in patients with diabetes is still debated. Studies in the literature are indeed controversial, possibly because these diets are generally poorly defined; this, together with the intrinsic complexity of dietary interventions, makes it difficult to compare results from different studies. Despite the evidence that reducing carbohydrates intake lowers body weight and, in patients with type 2 diabetes, improves glucose control, few data are available about sustainability, safety and efficacy in the long-term. In this review we explored the possible role of low-carb and ketogenic diets in the pathogenesis and management of type 2 diabetes and obesity. Furthermore, we also reviewed evidence of carbohydrates restriction in both pathogenesis of type 1 diabetes, through gut microbiota modification, and treatment of type 1 diabetes, addressing the legitimate concerns about the use of such diets in patients who are ketosis-prone and often have not completed their growth.
Type 1 Diabetes. Andreas Eenfeldt goes through the scientific and anecdotal evidence, and also what clinical experience tends to show, regarding the long-term effects of low carb. Follicular Thyroid Cancer. S7 Table Risk of bias assessment for Anderson et al. And perhaps not unsurprisingly, studies and experience have shown low-carb diets can be beneficial for people with type 1 diabetes.
Type 1 diabetes results when the body is no longer able to produce the hormone insulin. Type 1 diabetes is treated with insulin injections. The more carbohydrates a person with type 1 diabetes eats, the more insulin will need to be injected. And perhaps not unsurprisingly, studies and experience have shown low-carb diets can be beneficial for people with type 1 diabetes. Here is our guide to low-carb diets in type 1 diabetes. For other controversies related to low carb, see our full guide. Type 1 diabetes is an autoimmune disease that destroys the insulin-producing cells of the pancreas, known as beta cells. This ultimately leads to a complete loss of insulin production. This prompts the pancreas to produce more insulin, leading to high insulin levels known as hyperinsulinemia.